Structure
Function
Cytology
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2009
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November
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- Spine [1,3]
- Nuchal [1,2]
- Cranial [1,1]
- Pelvic [2,3]
- Abdominal [2,2]
- Thoracal [2,1]
- Endocrine, Paracrine, RES [3,3]
- Pituitary [3,2]
- Hypothalamus [3,1]
- Physiologically Active Proteins [4,3]
- Phospholipid Bilayer [4,2]
- Matrix Proteins [4,1]
- Biological Membranes [5,3]
- Reticulum [5,2]
- Endoplasm [5,1]
- Nucleolus [6,3]
- Nucleoplasm [6,2]
- Nucleus [6,1]
- Mitogenesis* [7,3]
- Kinetaxis* [7,2]
- Filaments [7,1]
- Protein [8,3]
- Peptide [8,2]
- Polypeptide [8,1]
- tRNA [9,3]
- mRNA [9,2]
- DNA [9,1]
- Medicine in a Nutshell
- Diagnosis
- Etiology
- Orders of Causality
- Biological Information Systems
- Beta Islet
- Jaundice
- Mevalonate Pathway
- Glycolysis
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- Delete*
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- Creativity->God->Artist->Beauty->Bayes->Tuber
- Tasks
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- Mutant Beta-Globulin
- Neurone Death
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November
(54)
Sulforafane
ReplyDeleteK5, 14
K17-> nrf2
K17-> filament structure
Filament function
Filament pathophysiology
Epidermilysis Bullisa:
BCC, SCC, MM: K5
Psoriasis:K17->Th1(Cytokine dominated) not Th2(Cytotoxic)
Anabolism <- Insulin <-|- Serine/Threo Kinase
ReplyDeleteGluca/FFA <- Insulin <- Tyrosine Kinase
Insulin Receptor <- Alpha/Beta Unit <- B = S/Th/TK
1' structure-> mRNA -> peptide bonds
ReplyDelete2' structure -> alpha-helix/beta-pleated -> hydrogen bonds
3' structure -> folding of 2' -> ionic/hydrophobic/sulfide bonds
Catalytic Protein Mutation -> Autosomal Recessive
ReplyDeleteNon-Catalytic Protein Mutation -> Autosomal Dominant
Merely A Rule of Thumb!!!