Thursday, November 19, 2009

Cranial [1,1]

Extracranial
Cranial
Intracranial

6 comments:

  1. HD gene -> Methylation of Ch.4 histones -> DNA Inactivity
    Neurotropic proteins not produced -> AD Complete Penetrance
    Progressive [exp{-H(t)}] Neurological deterioration

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  2. 0: prologue:
    cause
    mediator
    effect

    1: introduction
    nucleic acid
    protein
    functional-structure

    2: thesis
    nucleoplasm
    cytoplasm
    plasma-membrane

    3: conclusion
    semaphorins gene -> axon tragectory
    semaphorins -> growth + guidance molecule
    neuropilin -> dendrite/target/distribution

    0: epilogue
    cario/nucho/spinal
    cardio/pulmonary/mediastinal
    pelvic/abdominal/endocrine

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  3. pathophysiology not equal to [exp{-H(t)}]
    childhood (developmental) exposure
    genetic predisposition

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  4. Gene, KAL1 Xp22.3
    Protein, Anosmin-1
    Migration, Olfactory Placode

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  5. Gene, GGCX Active in Hepatocytes
    Gamma-Glutamyl Residues -> Gamma-Carboxy Glutamate
    Ca2+ Binding Sites for VII, IX, X -> Phospholipid Membrane

    ReplyDelete