HD gene -> Methylation of Ch.4 histones -> DNA InactivityNeurotropic proteins not produced -> AD Complete PenetranceProgressive [exp{-H(t)}] Neurological deterioration
0: prologue:causemediatoreffect1: introductionnucleic acidproteinfunctional-structure2: thesisnucleoplasmcytoplasmplasma-membrane3: conclusionsemaphorins gene -> axon tragectorysemaphorins -> growth + guidance moleculeneuropilin -> dendrite/target/distribution0: epiloguecario/nucho/spinalcardio/pulmonary/mediastinalpelvic/abdominal/endocrine
epilepsyschizophreniaautism
pathophysiology not equal to [exp{-H(t)}]childhood (developmental) exposuregenetic predisposition
Gene, KAL1 Xp22.3Protein, Anosmin-1Migration, Olfactory Placode
Gene, GGCX Active in Hepatocytes Gamma-Glutamyl Residues -> Gamma-Carboxy Glutamate Ca2+ Binding Sites for VII, IX, X -> Phospholipid Membrane
HD gene -> Methylation of Ch.4 histones -> DNA Inactivity
ReplyDeleteNeurotropic proteins not produced -> AD Complete Penetrance
Progressive [exp{-H(t)}] Neurological deterioration
0: prologue:
ReplyDeletecause
mediator
effect
1: introduction
nucleic acid
protein
functional-structure
2: thesis
nucleoplasm
cytoplasm
plasma-membrane
3: conclusion
semaphorins gene -> axon tragectory
semaphorins -> growth + guidance molecule
neuropilin -> dendrite/target/distribution
0: epilogue
cario/nucho/spinal
cardio/pulmonary/mediastinal
pelvic/abdominal/endocrine
epilepsy
ReplyDeleteschizophrenia
autism
pathophysiology not equal to [exp{-H(t)}]
ReplyDeletechildhood (developmental) exposure
genetic predisposition
Gene, KAL1 Xp22.3
ReplyDeleteProtein, Anosmin-1
Migration, Olfactory Placode
Gene, GGCX Active in Hepatocytes
ReplyDeleteGamma-Glutamyl Residues -> Gamma-Carboxy Glutamate
Ca2+ Binding Sites for VII, IX, X -> Phospholipid Membrane